A Mutation in the Latency-Related Gene of Bovine Herpesvirus 1 Disrupts the Latency Reactivation Cycle in Calves

Our studies revealed that a ␤ -catenin coactivator, high-mobility group AT– hook 1 protein (HMGA1), was read- ily detected in a subset of trigeminal ganglion neurons in

protein encoded by the bovine herpes virus 1 (BHV-1) latency related gene interacts with specific cellular regulatory proteins, including the CCAAT enhancer binding protein

Plasmids that expressed LR gene products promoted survival of monkey kidney (CV-1), human lung (IMR-90), or mouse neuroblastoma (neuro-2A) cells after induction of PCD.. Plasmids

In this report, we further characterize this LR transcrip- tionally active region (TAR) of the BHV-1 genome, including (i) fine mapping of the LR RNA in rabbit and bovine

Our results indicate that the LR promoter has strict orientation preferences in all cell types tested and that in bovine cells, promoter activity is stimulated by infection with

MCA were screened by the ELISA with purified BHV-1, purified BHV-1 nucleocapsid, BHV-1- infected cell lysate, herpes simplex virus type 2-infected cell.. lysate, and noninfected

Four of seven calves vaccinated under passive immunity became clearly BHV-1 seronegative by different serological tests, as did uninfected control calves after the disappearance

Infection of seven passively immunized young calves with a virulent strain of bovine herpesvirus type 1 (BHV-1) was performed to determine whether they could become seronegative