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Delayed Sequelae of Neonatal Respiratory Syncytial Virus Infection Are Dependent on Cells of the Innate Immune System

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Figure

FIG 1 Neonatal RSV infection leads to enhanced chemokine and proinflammatory cytokine release on subsequent RSV reinfection
FIG 2 Neonatal RSV infection leads to disease and increased innate effector cell recruitment on subsequent RSV reinfection
FIG 3 Disease during reinfection following primary neonatal (NN) RSV infection requires NK cells
FIG 4 Disease during reinfection following primary neonatal RSV infection requires macrophages

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