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ER stress regulates alkaline phosphatase gene expression in vascular smooth muscle cells via an ATF4 dependent mechanism

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Figure

Fig. 1 ALP expression and activity are ATF4-dependent in VSMCs. VSMCs were treated with 0.4 μg/ml TM or 0.2 μg/ml TG for 24 h
Fig. 3 ATF4 does not bind the ALP promoter in VSMCs. a ER stress-related transcription factor binding sites mapped to the ALP promoter

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