Epstein-Barr virus nuclear antigen 2 transactivates the long terminal repeat of human immunodeficiency virus type 1.

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The occurrence of these genotypes is shown as the number and percentage of specimens from which each genotype was detected as the dominant virus and the codominant (in

to give runoff transcripts, whereas P68-4 was generated by introducing a stop codon at amino acid 296, resulting in the formation of a truncated protein. The truncated proteins

The transactivator is generated by deletion of the C-terminal hydrophobic region III of MHBs but requires the presence of the N-terminal part of the S domain; insertion of a stop

(B) Gel retardation assay showing the specificities of TrpE (vector), TrpE-ZEBRA (Zl-245), and TrpE-Fos- GCN4 (Fos-GCN4) proteins for oligonucleotides containing the AP-1 and

The P genes of simian virus 5 (27), measles virus (2), Sendai virus (SEN) (29), human parainfluenza virus type 2 (PIV2) (21, 26), and mumps virus (8) were found to produce more than

Sequence-specific methylation of 5'-CCGG-3' sequences at positions +6, +24, and -215 leads to the inactivation of the late E2A promoter of adenovirus type 2 (Ad2) DNA, as

unedited mRNA was shown to encode the mumps virus cysteine-rich protein V, and mRNA transcripts containing two and four inserted G residues were translated to yield the mumps virus