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MicroRNA-Mediated Transformation by the Kaposi's Sarcoma-Associated Herpesvirus Kaposin Locus

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Figure

FIG 1 The miR-K10a miRNAs overcome contact inhibition in the absence ofKapA in NIH/3T3 cells
FIG 2 Continued transgene expression is required to maintain transformedstatus. (A) Schematic outlining the experimental setup used for secondaryfocus formation assays
FIG 3 Inhibition of mature miR-K10a/confirm inhibition of miR-K10a/�1 results in loss of the transformed phenotype in FS-transformed NIH 3T3 cells
FIG 5 Conservation of PAR-CLIP-identified miRNA binding sites betweenhuman and mouse. Sites were scored as conserved if at least a minimal matchto the miRNA was present in a two-way alignment between the human andmouse genomes (i.e., base pairing of nt 2 to
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