• No results found

Association of human immunodeficiency virus type 1 envelope glycoprotein with particles depends on interactions between the third variable and conserved regions of gp120.

N/A
N/A
Protected

Academic year: 2019

Share "Association of human immunodeficiency virus type 1 envelope glycoprotein with particles depends on interactions between the third variable and conserved regions of gp120."

Copied!
5
0
0

Loading.... (view fulltext now)

Full text

Loading

Figure

TABLE 1. Virus designations and relative infectivity
FIG. 3.gp120p66centrifugation.SDS-PAGEHIV-1Q-267/I-308,indicated.HeLaimmunoprecipitatedmixturemedium, Incorporation of envelope glycoproteins into virions

References

Related documents

Most of the mutant viruses also had normal Western blot (immunoblot) profiles, although three of the mutations resulted in reduced signals for IN relative to the wild type on

The relationship between the expression of ULli and resistance to HSV infection in Us11cl19 cells has not been defined, but the block to infection with wild-type HSV-1 was overcome

(43, 44) reported the role of Ca2+ in the in vitro assembly of murine polyomavirus recombinant VP1 capsomeres into capsid-like particles, and now we have reported a similar mechanism

Binding of trans-dominant mutant Rev protein of human immunodeficiency virus type 1 to the cis-acting Rev-responsive element does not effect the fate of viral mRNA.

with virion RNA plus liposome, replication of LDVwas seen in all cell types tested, i.e., macrophages from newborn and adult mice and cell lines from mouse and heterologous

AIDS patient sera precipitated both precursor and processed forms of the wild-type and AV1/2, AV3, and AV1/2/3 glyco- proteins from transfected COS-1 cells lysates..

Also, some of the AVif virions from CEM cells, which are not totally restrictive for the mutant, may be able to perform some reverse transcription in the target cell; completed

finding that protective MAbs could act at a variety of different stages in pathogenesis, including primary replica- tion, entry of a virus into the nervous system, and spread.