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Cytolysis by CCR5-Using Human Immunodeficiency Virus Type 1 Envelope Glycoproteins Is Dependent on Membrane Fusion and Can Be Inhibited by High Levels of CD4 Expression

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Academic year: 2019

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FIG. 3. Ability of Cf2Th-CD4/CCR5 and Cf2Th-CCR5 cells toCCR5 (black bars) and Cf2Th-CCR5 cells (white bars) to serve astarget cells for syncytium formation was examined
FIG. 4. Increase in cytotoxicity of a CD4-independent HIV-1 en-velope glycoprotein in CD4-expressing cells due to a decrease in CD4-
Fig. 6B and 6D. A 20-min exposure of the virus preparation to
FIG. 6. Generation of inactivated HIV-1 virions. (A) Virions that incorporated the HXBc2, HXBc2-P, 89.6, or KB9 envelope glycoproteins aswell as the F522Y fusion-defective variants of these envelope glycoproteins were made by cotransfecting the three plasm
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