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The helminth product, ES-62, protects against airway inflammation by resetting the Th cell phenotype

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Figure

Fig. 2. In vivo treatment with the phosphorylcholine-containing glycoprotein, ES-62, inhibits draining lymph node (DLN) cell antigen-specific Th2 cytokine production butnot antigen-specific proliferation ex vivo
Fig. 3. Exposure to the phosphorylcholine-containing glycoprotein, ES-62, in vivo does not modulate ovalbumin (OVA)-induced elevation of IgG1 and IgG2a but inhibits IgE
Fig. 5. Neutralising anti-IFNc antibodies prevent ES-62-mediated protection against cellular infiltration in murine ovalbumin (OVA)-induced airway inflammation.Differential neutrophil (A), macrophage (B), lymphocyte (C) and eosinophil (D) bronchoalveolar lav
Fig. 7. The phosphorylcholine-containing glycoprotein, ES-62, suppresses IL-17 responses in murine ovalbumin (OVA)-induced airway inflammation

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