• No results found

Molecular Determinants of Virulence, Cell Tropism, and Pathogenic Phenotype of Infectious Bursal Disease Virus

N/A
N/A
Protected

Academic year: 2019

Share "Molecular Determinants of Virulence, Cell Tropism, and Pathogenic Phenotype of Infectious Bursal Disease Virus"

Copied!
9
0
0

Loading.... (view fulltext now)

Full text

Loading

Figure

FIG. 1. Schematic presentation of various cDNA constructs of IM,D78, and GLS strains in order to generate plus-sense RNA transcripts
TABLE 1. Location of amino acid changes in VP2, VP4, VP3, and VP1 proteins of IBDV strains D78 and IM
TABLE 2. Generation of chimeric IBDVs after transfection withtranscripts derived from cloned cDNA of segments A and B in Vero
FIG. 2. Growth curves of rD78, rIMB, rIMVP4, rIMVP43, and rGLSVP2 IBDVs. Monolayers of CEF cells were infected with the indicatedviruses at a multiplicity of infection of 0.1 and harvested at the indicated time points, and infectious titers were determined by plaque assay.
+3

References

Related documents

In order to get more insights into the role of ER stress in the pathogenesis of diabetes mellitus and in the reduced total β -cell volume in male Munich Ins2 C95S mutant

We show that a differential introduced via the 61 st IV bit in Grain v1, leads to a dis- tinguisher for the keystream bit produced in the 105 th round in Grain v1, if certain

Experimental design: In this study, using stearic acid-g-chitosan oligosaccharide (CSO-SA) polymeric micelles as a drug-delivery system, OXA-loaded CSO-SA micelles (CSO-SA/OXA) were

Sequences in the pro- moter (Spl binding sites and the TATA motif) and enhancer (NF-KB binding sequence) regions constituted the most influ- ential positive control elements for

Alternatively, the membrane-proximal CD4 domains could Kinetics of rgpl2O binding to cells expressing CD4.CD8 or affect fusion by exerting their effect indirectly through the M

These results suggest that initiation of poliovirus RNA translation at the correct site requires the interaction of a factor(s) with the 5' UTR and that this factor(s) is more

Quantitative analysis of DNA profiles (Fig. 1) representing the frequency distribution of cells in Go+Gj, S, and G2+M phases of the cell cycle at various times after infection