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Figure

Figure 1: Fatp1 deletion decreased M F acyl-CoA synthetase (ACSL) activity and resulted in a metabolic shift from lipid oxidation to glycolysis that resulted in exacerbated CAM and reduced AAM activation
Figure 4: Overexpression of FATP1 in RAW264.7 M F s induced a substrate switch with enhanced lipid metabolism and reduced glucose metabolism resulting in blunted CAM-activation
Figure 5: Overexpression of FATP1 in RAW264.7 M F s decreased arachidonate and certain eicosanoid concentrations
Figure 6: FATP1 is a novel regulator of immunometabolism: Working Model: Fatty acid transport protein (FATP1) is necessary to maintain the alternatively activated macrophage (AAM) phenotype, limit oxidative stress, and to reduce high fat diet (HFD)-induced

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