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IgG glycan hydrolysis by EndoS inhibits experimental autoimmune encephalomyelitis

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Figure

Figure 1 EndoS treatment ameliorates clinical and histological MOG35-55-induced EAE. (A) A schematic representation of the experimentalsetup
Figure 2 Endoglycosidase treatment has a profound effect onfrom EndoS-treated WT mice (IgG glycan hydrolysis in vivo
Figure 4 EndoS inhibits peripheral complement activation in EAE mice.indicated time after immunization
Figure 5 High levels of C9 are detected in the spinal cord ofPBS-treated EAE mouse models, but absent from EAE micetreated with EndoS

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