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Expression of microRNA-30c via lentivirus vector inhibits the proliferation and enhances the sensitivity of highly aggressive ccRCC Caki-1 cells to anticancer agents

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Table 1 The concentrations of antitumor drugs used in this work
Figure 1 The endogenous expression of mir-30c is inversely associated with MTa-1 in clinical specimens.Notes: endogenous mrna level of MTa-1 (A, C) or rna level of mir-30c (B, D) was detected by qrT-Pcr in 10 nontumor (n1–n10), 10 primary ccrcc (c1–c10), a
Figure 2 expression of mir-30c is inversely associated with MTa-1 in kidney or ccrcc cell lines.Notes: The expression of MTa-1 (A, B) or mir-30c (C, D) in kidney nontumor cell lines hKc or heK293, and ccrcc cell lines caki-1 or 786-O was detected by quanti
Figure 3 mir-30c represses MTa-1 to inhibit the proliferation of caki-1 cells.Notes: (A) Western blot was used to examine the effect of miR-30c on MTA-1 expression
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