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Parkin deficiency prevents chronic ethanol-induced hepatic lipid accumulation through β-catenin accumulation

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Figure

Fig. 1 Alcoholic liver injury model induced by chronic binge-ethanol (EtOH) feeding in parkin KO mice.means ± SD; a The body weight and b daily food intake ofWT and parkin KO mice, which were fed control or ethanol-supplemented diet for 10 days
Fig. 2 Parkin KO mice show protection against ethanol-induced hepatic lipid accumulation.(SREBP-1c and CD36) and lipid oxidation (AOX and MCAD) in the livers of pair- or ethanol-fed WT mice and parkin KO mice.means ± SD;parkin KO mice were stained with hem
Fig. 3 The loss of parkin decreases ethanol-induced lipid accumulation and regulates expression of genes involved in lipid metabolism in mousehepatic cells
Fig. 4 Ethanol-fed parkin KO mice show β-catenin accumulation in the liver. a Liver sections were triple-stained with β-catenin antibody (Alexa488; green, arrowahead), parkin antibody (Alexa 568; red), and DAPI (nuclear counterstain; blue), and images were
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