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Dap160/intersectin binds and activates aPKC to regulate cell polarity and cell cycle progression

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Figure

Fig. 1. Dap160 interacts with aPKC. (protein coupled to glutathione S-transferase (GST) beads can bind in vitro produced aPKC protein (arrowhead)
Fig. 2. Dap160 co-localizes with aPKC inneuroblasts. (A-L) Neuroblasts co-stained forDap160 (A-F) and aPKC (G-L); merged imagesbelow
Fig. 4. Dap160 positively regulates neuroblastpool size. (A) Neuroblast numbers scored wild-type
Fig. 5. Dap160 is required for neuroblast cell cycle progression. Wild-type, AO interval of 17.84±4.52 minutes; dap160 mutant and aPKC mutant neuroblasts imaged withJupiter::GFP from nuclear envelope breakdown (NEBD) to anaphase onset (AO)

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