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Mary Bradbury, PharmD, BCPS Clinical Pharmacy Specialist, Cardiac Surgery September 18, 2012 [email protected]

This presentation will discuss unlabeled and investigational use of products

The author has no disclosures

(2)

2.5 million Americans live with atrial fibrillation (afib)

Afib-associated morbidity and mortality

Annual incidence of stroke in patients with afib: 5%

Risk of stroke increases with age

Reprinted from AF Stat™: A Call to Action for Atrial Fibrillation; Accessed via www.AFStat.com

2010 AHA/ASA guidelines for the primary prevention of stroke

2010 AHA/ASA guidelines for prevention of stroke in patients with ischemic stroke or TIA 2012 ACCP evidence-based clinical practice guidelines: antithrombotic therapy for atrial fibrillation and prevention of thrombosis, 9

th

Ed.

2012 AHA/ASA advisory on oral

antithrombotic agents for the prevention of

stroke in nonvalvular atrial fibrillation

(3)

Which patients with afib need stroke prevention therapy?

Risk stratification: CHADS2 Score ≥ 2

Risk Factor Points

Congestive heart failure 1

Hypertension 1

Age ≥ 75 years 1

Diabetes 1

Stroke or TIA 2

CHADS2 score

Risk group

Recommendation (Level of evidence)

Annual absolute risk

ASA VKA

0 Low Nothing or ASA only (2B) 0.6% 0.3%

1 Moderate Anticoagulation (1B) 1.7% 0.8%

2 High Anticoagulation (1A) 3.6% 1.7%

3-6 High Anticoagulation (1A) 7.6% 3.6%

ASA: Aspirin; VKA: Vitamin K antagonist

Antithrombotic therapy and prevention of thrombosis, 9thEd: ACCP Guidelines

(4)
(5)

Safety Efficacy

(6)

FDA approved October 2010

150 mg PO BID for CrCL > 30 mL/min

75 mg PO BID for CrCL 15-30 mL/min (not studied) Unique mechanism of action: direct thrombin inhibitor (argatroban, bivalirudin)

150 mg dose demonstrated superior efficacy and similar bleeding rates vs. warfarin (RELY)

Mean CHADS2 score: 2.1

Connolly SJ. N Engl J Med 2009;361(9):1139-1151.

Furie KL. Stroke 1012;43:00-00.

(7)

Event Warfarin (INR 2-3)

Dabigatran 150

mg BID P Value

Stroke

Hemorrhagic Ischemic

1.69%

0.38%

1.20%

1.11%

0.10%

0.92%

< 0.001

< 0.001 0.03 Major bleeding

Intracranial Life threatening Gastrointestinal

3.36%

0.74%

1.80%

1.02%

3.11%

0.30%

1.45%

1.51%

0.31

< 0.001 0.04

< 0.001

Dyspepsia 5.8% 11.3% < 0.001

Myocardial infarction (MI) 0.53% 0.74% 0.048

All results reported as % per year Connolly SJ. N Engl J Med 2009;361(9):1139-1151.

RELY substudy: platelet function test No difference in platelet activation between warfarin and dabigatran groups

Meta-analysis supports increase risk of MI Risk of MI/ACS: 1.19% dabigatran vs. 0.79%

comparator (P = 0.03)

Uchino K. Arch Intern Med 2012;172(5):397-402.Ferreira J. Thrombosis 2012 Uchino K. Arch Intern Med 2012

(8)

ACCP 9

th

Ed. (January 2012)

For those with atrial fibrillation and risk score favoring anticoagulation, dabigatran

recommended rather than VKA therapy (2B) with the exception of patients with the following:

▪ Mitral stenosis

▪ Active coronary artery disease (CAD) or history of CAD

You JJ. Chest 2012;141(2)Suppl:e531s-e575s.

AHA/ASA Advisory (August 2012)

“Dabigatran Is useful as an alterative to warfarin…

in patients who do not have a prosthetic heart valve or hemodynamically significant valve disease,

severe renal failure, or advanced liver disease (1B).”

Furie KL. Stroke 2012;43:00-00.

(9)

Lack of monitoring does not always prove beneficial

Renal function must be monitored closely

Lack of reversal agent

New Zealand experience identifies 4 items likely contributing to bleeding complications:

– Prescriber error (renal impairment, bridging off warfarin) – Acute changes in renal function

– Patient age and weight, especially > 80 years and < 60 kg – Complications secondary to prolonged half-life and lack of

reversal

Harper P. N Engl J Med 2012;366;9:864-866.

(10)

FDA approved November 2011

20 mg PO daily with meal for CrCL > 50 mL/min 15 mg PO daily with meal for CrCL 15-50 mL/min Mechanism of action: Inhibitor of factor Xa Similar efficacy and similar bleeding rates vs. warfarin (ROCKET AF)

Mean CHADS2 score: 3.5

Patel MR. N Engl J Med 2011;365;10: 883-891.

Event Warfarin

(n=7125)

Rivaroxaban

(n=7111) P Value

Stroke

Hemorrhagic

2.2%

1.2%

1.7%

0.8%

< 0.001*

0.02

Major bleeding 5.4% 5.6% 0.31

Myocardial

infarction 1.1% 0.9% 0.12

All results reported as % per year

*Results reported for per protocol analysis (n=7004 for warfarin and n=6958 for Rivaroxaban) and as non-inferiority

Patel MR. N Engl J Med 2011;365;10: 883-891.

(11)

ACCP Antithrombotic guidelines: no recommendation to date

AHA/ASA

In patients with CrCL > 50 mL/min and afib at high risk of stroke (CHADS2 ≥ 2), rivaroxaban is a

reasonable alternative to warfarin (IIa)

▪ The safety and efficacy of the renally adjusted dose has not been clearly established (IIb)

Lack of monitoring does not always prove beneficial

Safety and efficacy with renal and hepatic dysfunction not clearly established

Lack of reversal agent

Lack of post-marketing surveillance

Harper P. N Engl J Med 2012;366;9:864-866.

(12)

Not yet FDA approved

Studied as 5 mg PO BID

2.5 mg PO BID suggested in patients with at least two of the following:

–Age ≥ 80 years, weight ≤ 60 kg, creatinine ≥ 1.5 mg/dL

Mechanism of action: Inhibitor of factor Xa

Superior efficacy and safety vs. warfarin (ARISTOTLE)

Mean CHADS2 score: 2.1

Granger CB. N Engl J Med 2011;365:981-992.

Event Warfarin

(n=9081)

Apixaban

(n=9120) P Value Stroke

Hemorrhagic Ischemic

1.60%

0.47%

1.05%

1.27%

0.24%

0.97%

0.01

< 0.001 0.42 Major bleeding

Intracranial Gastrointestinal

3.09%

0.80%

0.86%

2.13%

0.33%

0.76%

< 0.001

< 0.001 0.37 Myocardial

infarction 0.61% 0.53% 0.37

All-cause mortality 3.94% 3.52% 0.047

All results reported as % per year

Granger CB. N Engl J Med 2011;365:981-992.

(13)

ACCP Antithrombotic guidelines: no recommendation to date

AHA/ASA

If CHADS2 score ≥ 1 and VKA unsuitable, apixaban is reasonable alternative to ASA therapy (I)

If CHADS2 score ≥ 2, apixaban is reasonable alternative to VKA therapy (I)

▪ If either of above is true and patient has > 1 risk factor for decreased drug clearance (age, weight, creatinine), consider lower dose alternative (IIb)

▪ Avoid apixaban if CrCL < 25 mL/min (Class III)

Never bridge No monitoring

Consider all factors that may lead to drug accumulation

Route of elimination: renal +/- hepatic Drug interactions due to CYP3A4 or P- glycoprotein

Advanced age or low body weight

(14)

Warfarin Class I

• INR goal 2-3

Dabigatran Class I

• Dose adjust based on CrCL and drug interactions

• Level of evidence lower for renally adjusted dose, avoid if CrCL < 15 mL/min

Apixaban Class I

• Dose adjust based on age, weight, creatinine*

• Level of evidence lower for renally adjusted dose, avoid if CrCL < 25 mL/min

Rivaroxaban Class IIa

• Dose adjust based on CrCL

• Avoid if CrCL < 15 mL/min or moderate-severe hepatic dysfunction

*Drug/dosing not currently FDA approved

(15)

Contraindicated with therapeutic warfarin 2007 Stroke Guidelines recommend INR ≤ 1.7 Dabigatran: not recommended

Normal aPTT and thrombin time suggest complete elimination of dabigatran

Rivaroxaban: not recommended

Normal PT may suggest complete elimination of rivaroxaban

Apixaban: not recommended

Adams HP. Stroke 2007;38:1655-1711

MINIMIZE ABSORPTION

Activated charcoal if ingestion occurred within 2 hours MINIMIZE ABSORPTION

Activated charcoal if ingestion occurred within 2 hours

MAXIMIZE RENAL ELIMINATION Assure adequate hydration & diuresis

Hemodialysis: 68% of drug is removed in a 4-hour dialysis session MAXIMIZE RENAL ELIMINATION

Assure adequate hydration & diuresis

Hemodialysis: 68% of drug is removed in a 4-hour dialysis session

BLOOD PRODUCTS FOR HEMODYNAMIC SUPPORT FFP will not reverse the coagulopathy of dabigatran BLOOD PRODUCTS FOR HEMODYNAMIC SUPPORT

FFP will not reverse the coagulopathy of dabigatran

OTHER AGENTS

Non-specific reversal agents? Activated PCC, rFVIIa Monoclonal antibody

OTHER AGENTS

Non-specific reversal agents? Activated PCC, rFVIIa Monoclonal antibody

PCC: Prothrombin complex concentrate, rFVIIa: recombinant activated factor VII

(16)

MINIMIZE ABSORPTION

Activated charcoal if ingestion occurred within 2 hours MINIMIZE ABSORPTION

Activated charcoal if ingestion occurred within 2 hours

BLOOD PRODUCTS FOR HEMODYNAMIC SUPPORT FFP will not reverse the coagulopathy of dabigatran BLOOD PRODUCTS FOR HEMODYNAMIC SUPPORT

FFP will not reverse the coagulopathy of dabigatran

OTHER AGENTS Non-specific reversal agents?

Activated PCC, PCC, rFVIIa OTHER AGENTS Non-specific reversal agents?

Activated PCC, PCC, rFVIIa

PCC: Prothrombin complex concentrate, rFVIIa: recombinant activated factor VII

Mary Bradbury, PharmD, BCPS Clinical Pharmacy Specialist, Cardiac Surgery September 18, 2012 [email protected]

References

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