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RIP3 promotes colitis-associated colorectal cancer by controlling tumor cell proliferation and CXCL1-induced immune suppression

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Figure

Figure 1. RIP3 expression is upregulated in AOM/DSS tumors and human colorectal carcinoma (CRC)
Figure 3. RIP3 promotes epithelial cell proliferation and tumor growth without affecting apoptosis
Figure 5. RIP3 deletion enhances the immunogenicity of the tumor microenvironment during CAC
Figure 6. Deletion of RIP3 reduces CXCL1 expression. (A-B) Differences in gene expression between tumors and normal colon tissues derived from WT and Rip3were used
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