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CCT6A suppresses SMAD2 and promotes prometastatic TGF β signaling

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Figure

Figure 3. SMAD2 mediates a tumor-suppressive transcriptional program, whereas SMAD3 mediates a pro-cell survival transcriptional program, in NSCLC cells
Figure 4. CCT6A specifically interacts with SMAD2 and suppresses the SMAD2-mediated transcriptional program
Figure 6. CCT6A mediates TGF-β–promoted metastasis of NSCLC cells. (A–D) Mice were intravenously injected via tail vein with the indicated NSCLC cells

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