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Dual-targeting of aberrant glucose metabolism in glioblastoma

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Figure

Table 1 Cell viabilities of GBM cells and non-cancerous cells after treatments with PENAO, DCA and combination
Figure 3 The oxidative stress plays a prominent role in the cytotoxicity of PENAO-DCA combination
Figure 4 DCA treatment inhibits MRP1 expression, thereby increasing the cytosolic accumulation of PENAO in GBM cells
Figure 5 DCA promotes oxidative phosphorylation through inhibiting glycolysis and attenuates PENAO-induced acid production.and the combination

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