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Type I IFNs mediate development of postinfluenza bacterial pneumonia in mice

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Figure

Figure 1Primary influenza pneumonia enhances sensitivity to secondary pneumococcal infection
Figure 2Influenza-infected
Figure 3Lung homogenates from higher levels of KC and Mip2. Produc-tion of KC and Mip2 in response to secondary pneumococcal infection, as determined by ELISA analysis of Ifnarenza infection
Figure 4Type I IFNs inhibit production of
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