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p47phox is required for atherosclerotic lesion progression in ApoE–/– mice

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Figure

Figure 1(a) RT-PCR of p47phox and β-actin in wild-type SMCs after throm-bin stimulation (2 U/ml)
Figure 3Intracellular ROS production is decreased in p47The reduction in aconitase activity is reversed by pretreatment withpolyethylene glycol–conjugated superoxide dismutase (PEG-SOD).(phox–/– SMCs com-pared with wild-type SMCs
Figure 4
Table 1
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