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ER stress and Rho kinase activation underlie the vasculopathy of CADASIL

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Figure

Figure 1. Reduced vasodilation and vasoreactivity in CADASIL arteries. Assessment of vascular functional respons-es in small arteries obtained from CADASIL and control subjects by wire myography
Figure 2. Vascular remodeling and altered mechanical properties in CADASIL arteries. Vascular structure and mechanical proper-ties were assessed in pressurized small arteries obtained from CADASIL and control subjects
Figure 3. Mutant NOTCH3 gain of func-tion in CADASIL VSMCs. (A) Represen-
Figure 4. Increased RhoA/Rho kinase activity and blunted [Ca P < 0.01 versus control; #P < 0.05 versus vehicle plus CADASIL.CADASIL and control VSMCs in the absence and presence of a tative immunoblots
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