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Original Article Resveratrol protects human renal proximal tubular cells from high glucose and hypoxia/ reoxygenation induced injury via inhibiting p38-MAPK and thioredoxin-interacting protein (TXNIP) pathways

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Academic year: 2020

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Figure

Figure 1. RES treatment alleviated HG-induced H/R injury. Cell viability was determined by CCK-8 assay after treat-ment of HK-2 cells with different concentrations of resveratrol for 72 hours (A)
Figure 2. RES restrained oxidative stress injury in HK-2 cells with the model of HG combined H/R (HH/R)
Figure 4. Effect of blockage of p38-MAPK on HG-induced H/R injury and protective of RES treatment
Figure 5. Effect of blockage of p38-MAPK on the oxidative stress and the level of apoptosis as well as TNF-α with HG-induced H/R, intracellular ROS (A) was detected by flow cytometry, MDA (B) contents and SOD (C) contents were detected by commercially-avai
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