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Cyclic MOG₃₅–₅₅ ameliorates clinical and neuropathological features of experimental autoimmune encephalomyelitis

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Figure

Fig. 1. Clinical course and outcome parameters of EAE with or without c-MOGin dot plots show means.immunized only with the c-MOGdisease onset
Fig. 2. (A) Demyelinating, (B) inflammatory and (C,D) axonopathic processes of control and +cMOG groups during the acute phase of EAE, in spinal cords
Fig. 3. (A) Demyelinating, (B) inflammatory and (C,D) axonopathic processes of control and +cMOG groups during the chronic phase of EAE, in spinal cords
Table 1Highest ranked peptides using the consensus approach for prediction of peptide binding affinity
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