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Epstein-Barr Virus Nuclear Antigen 3C Activates the Latent Membrane Protein 1 Promoter in the Presence of Epstein-Barr Virus Nuclear Antigen 2 through Sequences Encompassing an Spi-1/Spi-B Binding Site

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FIG. 1. J�transfected with either empty pSG5 expression vector (control) or pSG5-EBNA-2 in the presence or absence of pSG5-EBNA-3C
FIG. 2. EBNA-3C activated the LMP-1 promoter in the presence of endog-enous levels of EBNA-2 in an EBV-positive BL cell line
FIG. 8. Transcriptionally active EBNA-2 is required for EBNA-3C-mediatedactivation of the LMP-1 promoter
FIG. 11. EBNA-3C binds to the ets domain of Spi-1 and Spi-B. GST fusionproteins that contained full-length Spi-1 (GST-Spi-1), the amino-terminal acti-

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