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Transcriptome profiling of monocytes from XLA patients revealed the innate immune function dysregulation due to the BTK gene expression deficiency

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Academic year: 2020

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Figure

Table 1. Clinical and immunological data of the XLA patients. N/A, not available. UD, undetectable
Figure 1. Hierarchical clustering of DE protein-coding genes and DE lncRNAs in primary monocytes of XLA patients compared to healthy subjects
Table 2. The upregulated genes involved in Oxidative Phosphorylation pathway in primary monocytes of the XLA patients compared to healthy subjects.
Table 3. The downregulated genes involved in immune-related pathways in primary monocytes of XLA patients compared to healthy subjects.
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