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Original Article Effects of sulforaphane on TNF-α-induced proinflammatory cytokine production and reactive oxygen species generation in mouse brain endothelial cells

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Figure

Figure 1. Reducing effect of SFN on TNF-α-induced proinflammatory cytokine production in bEnd.3 cells
Figure 2. Decreasing ef-fect of SFN on TNF-α-
Figure 3. Increasing effect of SFN on HO-1 expres-sion in TNF-α-treated bEnd.3 cells. The mRNA (A) and protein (B) expression levels of HO-1 in TNF-α-induced bEnd.3 cells, with or without SFN treat-ment, were detected with real-time PCR and West-ern blot a
Figure 4. Involvement of HO-1 in the effects of SFN on TNF-α-treated bEnd.3 cells. The TNF-α-treated bEnd.3 cells were pre-treated with the HO-1 activator CoPP (0.3 μmol/L) and inhibitor ZnPP (0.1 µmol/L), respectively, together with SFN (30 μg/mL)

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