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β Cell–intrinsic β arrestin 1 signaling enhances sulfonylurea induced insulin secretion

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Figure

Figure 2). Likewise, both groups of mice displayed similar blood
Figure 3. Control islets, but not β-barr1-KO islets, show greatly reduced glibenclamide-induced insulin
Figure 4. Glibenclamide promotes the interaction of Barr1 with Epac2 and stimulates Rap1 activation in a Barr1-dependent fashion

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