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Strategy to enhance the therapeutic effect of doxorubicin in human hepatocellular carcinoma by selenocystine, a synergistic agent that regulates the ROS-mediated signaling

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Academic year: 2020

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Figure

Figure 1: SeC enhances DOX-induced growth inhibition against HepG2 cells through enhanced intracellular uptake of DOX
Figure 2: Apoptosis and cell cycle arrest induced by
Figure 3: Contribution of ERK and AKT pathways to the combined treatment-induced growth inhibition against HepG2 cells
Figure 4: SeC synergizes DOX-induced DNA damage through ROS overproduction. A. SeC enhances DOX-induced ROS accumulation
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