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Relationship of ERK1_2 phosphorylation to D1 dopamine receptor activation, behavioral sensitization, and structural plasticity in the neonate 6-hydroxydopamine-lesioned rat

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Academic year: 2020

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Figure

Figure 1-1: ERK1/2 integration of diverse cell-surface signaling mechanisms.  AC,  adenylyl cyclase; PLC, phospholypase C; DAG, diacylglycerol; CaM, calmodulin;
Figure 1-2: Classic model of the basal ganglia circuit in (A) normal rats, (B)  neonate-lesioned rats, and (C) D 1  agonist-treated neonate-lesioned rats
Figure 1-3: Simplified model of the ‘motive’ circuit in (A) normal rats and (B) D 1
Fig. 2-1: Immunohistochemistry for tyrosine hydroxylase. Immunohistochemistry for  tyrosine hydroxylase (1:5000; Calbiochem) in coronal sections representing the striatum,  accumbens, and mPFC in sham-lesioned and neonate 6-OHDA-lesioned (Lesioned) adult
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