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Insights into 4E-BP1 and p53 mediated regulation of accelerated cell senescence

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Figure

figure 1: the p53 senescence pathway. Several triggers such as overexpression of oncogenes, telomere dysfunction or attrition, and genotoxic stress, which includes discodermolide treatment, promote the increased activity of p53
figure 2: Discodermolide-induced accelerated cell senescence. Senescence-associated β-galactosidase activity in A549 lung carcinoma cells treated with control (DMSO) or, an IC50 concentration of discodermolide for 6 days
Figure 3: A simplified schematic of the PI3K/Akt/mTORC1 pathway. Growth factors or hormones can stimulate phosphatidylinositol 3-kinase (PI3K) signaling

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