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Hypoxia suppresses cylindromatosis (CYLD) expression to promote inflammation in glioblastoma: possible link to acquired resistance to anti-VEGF therapy

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Academic year: 2020

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Figure

Figure 1: Effects of hypoxia on expression of CYLD and proinflammatory cytokines in GBM cells
Figure 2: Overexpression of CYLD inhibited hypoxia-induced inflammatory responses. (A) Western blotting to determine the relative amounts of FLAG-tagged exogenous (exo) CYLD protein versus endogenous (endo) protein
Figure 3: Effects of chronic bevacizumab treatment on histology of GBM xenografts. (A) Sections of U87MG-vector and U87MG-CYLD tumors treated with PBS or bevacizumab were stained with an antibody against the endothelial cell surface marker CD31 (left panel
Figure 4: Effects of CYLD overexpression on expression of inflammatory cytokines and survival of mice after bevacizumab treatment

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