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Original Article Total alkaloids of tripterygium hypoglaucum (levl.) Hutch (THHta) attenuates high glucose-induced podocyte injury via mediating miR-29a/PTEN axis

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Academic year: 2020

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Figure

Figure 1. Glucose inhibits podocyte proliferation, promotes levels of ROS and apoptosis
Figure 2. THHta attenuates podocyte injury induced by HG. Podocytes were treated with 30 mM glucose, and then incubated with 1.25, 2.5, or 5 μg/mL THHta for 48 h
Figure 3. THHta modulates miR-29a expression in HG-treated podocytes. A. Hierarchical clustering image showing the significant gene expression profiles of differentially expressed miRNAs in HG-treated podocytes which stimulated with or without THHta
Figure 4. THHta exerts the protective effects in HG-induced podocyte injury by upregulating miR-29a
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