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Unique CD14+ intestinal macrophages contribute to the pathogenesis of Crohn disease via IL 23/IFN γ axis

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Figure

Figure 1CD14-expressing cells were increased in the intestinal mucosa of patients with IBD
Figure 2CD14+CD33+ cells in the human intestinal LP revealed unique phenotypes and produced larger amounts of proinflammatory cytokines than CD14–
Figure 3CD14+ intestinal macrophages from patients with CD produced abundant levels of IL-23 and TNF-α in response to commensal bacteria antigen stimulation
Figure 4Commensal bacteria induced IFN-γ but not IL-17 by LPMCs via IL-23 produced by CD14+ intestinal macrophages
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