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Changes in alternative splicing of human and mouse genes are accompanied by faster evolution of constitutive exons

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Figure

Table 2Medians and Standard Deviations ofOrthologous Comparisons for Genes with AlternativeSplicing (AS) Conserved Between Humans and MouseCompared to Genes with Genome-Specific Alternative Ka and Ka/Ks fromSplicing in Humans or Mouse
Table 3
Table 4Mouse EST Coverage (Expressed As the MedianPercentage of Human EST Coverage for Each Gene)for Genes Having Alternatively Spliced (AS) Exons inHumans But for Which the Homologous Mouse ExonIs Either Consistently Skipped (Mouse Skip) orConsistently Included (Mouse Inclusion)

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