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CCR2 modulates inflammatory and metabolic effects of high fat feeding

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Figure 2Body mass of Ccr2–/– mice. (A) C57BL/6J Ccr2+/+ (black symbols) and Ccr2–/– (gray symbols) mice were fed a low-fat (triangles) or a high-fat diet (squares) for 24 weeks
Figure 3Insulin sensitivity in obese Ccr2–/– and obese Ccr2+/+ mice. Fasting plasma insulin (A) and blood glucose concentra-tions (B) were measured in lean Ccr2+/+ (black bars) and Ccr2–/– (gray bars) mice and mice of both genotypes made obese following 20
Figure 4Glucose homeostasis in obese Ccr2–/– and Ccr2+/+ mice. (A) The response of fast-ed obese Ccr2+/+ (black circles) and Ccr2–/– (gray squares) mice following a single intraperitoneal injection of insulin (1.5 U/kg) was monitored by serially measuring
Figure 6Expression of genes involved in inflammation. Quantitative RT-PCR (dark gray bars) mice with body mass greater than 40 g were studied, as were lean bars)
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