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Menin determines K RAS proliferative outputs in endocrine cells

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Figure

Figure 1. Pancreatic endocrine mass in Kras-het mice. (A and B) Schematic in A illustrates the relationship between K-RAS-GTP binding and intracellu-lar signaling; schematic in B illustrates the conversion of the transcriptionally silent KrasG12D–knock-in
Figure 2. Endocrine neogenesis and proliferation in Kras-het mice. (A) Diagram shows the time course of endocrine neogenesis and proliferation in the mouse pancreas (13)
Figure 3. K-RAS signaling suppresses endocrine cell growth. (of Ins2-CreA and B) Projection images of the dorsal pancreas were collected by whole-mount confocal microscopy from E13.5 mouse embryos of the indicated genotypes and immunostained for E-cadherin
Figure 4. Endocrine proliferation in menin-sensi-tive tissues. (A and B) Ventral views of microdis-sected tissue from P5 neonates show the trachea (Tr), thyroid (Th), and parathyroid (Pa, outlined by intercalated black melanocytes)
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