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Original Article Emodin inhibited ozone stress-induced airway hyperresponsiveness by regulating epithelial protection mechanisms against injury

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Figure 1. AR was assayed by Buxco system (n=6). Ozone resulted in increased AR to methacholine (Mch) and Emodin (50 mg/kg) abolished the in-creased AR induced by ozone (**P<0.01 versus con-trol; ##P<0.01 versus Ozone).
Figure 2. Lung histological pathology was assayed by HE
Figure 4. Cytotoxicity of Emodin on BECs (n=3, **P<0.01 vs. 0 µg/mL). The sig-
Figure 5. Effects of Emodin on TGF-β and PGE2 production from BECs (n=6). Ozone promoted the production of TGF-β and inhibited the production of PEG2

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