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3′,4′ dihydroxyflavonol ameliorates endoplasmic reticulum stress induced apoptosis and endothelial dysfunction in mice

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Academic year: 2020

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Figure

Figure 1. Chronic treatment with DiOHF normalizes systolic blood pressure, body weight and improves Test, endothelial function in mice treated with tunicamycin
Table 1. Agonist sensitivity (pEC-independent relaxations to acetylcholine (ACh) and sodium nitroprusside (SNP) in aortic rings isolated from all treatment groups
Figure 3. DiOHF treatment for two weeks increased total nitrate/nitrite level. The decreased total nitrite/nitrate levels in ER stress-induced mouse aortae were significantly reversed by the co-treatment of DiOHF and TUDCA
Figure 4. Chronic treatment with DiOHF alleviates ER stress, oxidative stress and apoptosis in mouse  vs control, Bonferroni’s Multiple Comparison Test
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