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Potentiation of imatinib by cilostazol in sensitive and resistant gastrointestinal stromal tumor cell lines involves YAP inhibition

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Academic year: 2020

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Figure 1: Cilostazol, a PDE3 inhibitor synergized with imatinib to reduce GIST48 cells viability
Figure 2: Differential expression of transcription factors and differentiation markers in GIST882 and GIST48 cells.
Figure 3: YAP expression in human GIST primary tumors. A) Immunohistochemistry for KIT-ir and YAP-ir in three different human GIST primary tumors of stomach, small intestine and colon, respectively
Figure 4: Cilostazol, but not imatinib, leads to YAP nuclear exclusion in imatinib-sensitive GIST882 and imatinib- imatinib-resistant GIST48 cells
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