Long non-coding RNA TUG1 promotes cell progression in hepatocellular carcinoma via regulating miR-216b-5p/DLX2 axis
Full text
Figure
Related documents
The mRNA and protein levels of IGF-1R were suppressed in Hep3B cells after miR-944 overexpression, whereas downregula- tion of miR-944 led to the opposite results in Huh7 cells
(A) qRT-PCR showing the expression of TUG1 mRNA in LoVo/si cells was significantly decreased compared with control cells (**P < 0.01); (B) qRT-PCR showing the expression of TUG1
Compared with the negative control, the level of MAP2K1 was conspicuously reduced by the knockdown of PART1 in SK-HEP-1 and Hep3B cells, however, MAP2K1 expression was
Additionally, Western blot assays showed that compared with that of the control group, the expression of HMGA2 on protein level was signi fi cantly increased after overexpression of
The expression of FOSL2 was regulated by miR-423-5p at the mRNA and protein levels after transfection of miR-423-5p mimics, miR-423-5p inhibitors, or negative control in 5-8F and
Figure 3 Paeonol down-regulated miR-21-5p level, and silencing of miR-21-5p inhibited cell viability, migration, invasion and promoted apoptosis in Hep3B and Huh-7 cells. (A)
Figure 7 FlOT1 overexpression attenuated the inhibitory effect on cell proliferation and the promoted effects on cell apoptosis, autophagy in 786-0 and A498 cells caused by miR-31-5p.
TDO mRNA and protein expression were detected in 5 different human HCC cell lines compared to the normal liver cell line Lo2.. We observed higher TDO expression in the Huh7 cell