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Original Article Upregulation of miR-18 controls the progression of hepatocellular carcinoma by modulating TGF-beta signaling

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Academic year: 2020

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Figure

Figure 1. Decreased miR-18 expression in HCC tissues and cell lines. A. Compared with normal para-carcinoma tissue, the level of miR-18 was significantly upregulated in HCC tissues
Figure 2. Inhibition of miR-18 prompts the process of EMT and cell migration. A. The cells demonstrated an elon-gated and spindle-shaped morphology after transfection with miR-18 inhibitor for 48 h
Figure 3. Smad2 is a target gene of miR-18. A. Schematic analysis showed the binding sites of miR-18 in the 3’UTR of Smad2
Figure 4. Overexpression of miR-18 abolished TGF-β-induced EMT pro-cesses in HepG2 cells

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