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Original Article Toll-like receptor 3 (TLR3) functions as a pivotal target in latent membrane protein 1 (LMP1)-mediated nasopharyngeal carcinoma cell proliferation

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Academic year: 2020

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Figure

Figure 1. LMP1 rescued the inhibited NPC cell proliferation caused by poly (I:C) treatment
Figure 2. LMP1 promoted TLR3 expression. A. LMP1 overexpression in CNE1 and HNE2 substantially increased TLR3 production
Figure 3. LMP1-transfection and poly (I:C) stimulation were able to induce TLR3 expression and NF-κB p65 activa-tion (A)
Figure 4. LMP1 rescued the impaired NF-κB p65 activation and subsequent pro-inflammatory gene production in the TLR3-knockdown cells
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