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Myeloid-derived suppressor cells contribute to A2B adenosine receptor-induced VEGF production and angiogenesis in a mouse melanoma model

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Academic year: 2020

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Figure

Figure 1: A2B receptor promotes tumor growth by altering the number of tumor-infiltrated immune cells
Figure 2: Increased tumor angiogenesis in melanoma-bearing mice treated with Bay60-6583 compared with control mice
Figure 3: CD11b+Gr1+ cells produce VEGF, that contributes to tumor angiogenesis. A. Immunofluorescence staining of Gr1+ cells with VEGF in melanoma tissue sections
Figure 4: STAT3 activation is enhanced in melanoma tissues of mice treated with Bay60-6583
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