M2-like macrophages
Ixmyelocel-T, an expanded multicellular therapy, contains a unique population of M2-like macrophages
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Tumor cell-released autophagosomes (TRAPs) promote immunosuppression through induction of M2-like macrophages with increased expression of PD-L1
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TLR2 stimulation impairs anti inflammatory activity of M2 like macrophages, generating a chimeric M1/M2 phenotype
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Dysregulated heme oxygenase 1low M2 like macrophages augment lupus nephritis via Bach1 induced by type I interferons
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Activation of mesenchymal stem cells by macrophages promotes tumor progression through immune suppressive effects
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Macrophage M2 polarization induced by exosomes from adipose-derived stem cells contributes to the exosomal proangiogenic effect on mouse ischemic hindlimb
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Original Article Expression of stabilin-1 in M2 macrophages in human granulomatous disease and melanocytic lesions
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Metformin prevents cancer metastasis by inhibiting M2-like polarization of tumor associated macrophages
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Macrophages inhibit human osteosarcoma cell growth after activation with the bacterial cell wall derivative liposomal muramyl tripeptide in combination with interferon-γ
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Roles of M1 and M2 Macrophages in Herpes Simplex Virus 1 Infectivity
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Macrophage polarization in periodontal ligament stem cells enhanced periodontal regeneration
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The macrophages in rheumatic diseases
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Clinical significance of macrophage phenotypes in cardiovascular disease
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Role of innate and adaptive immunity in obesity associated metabolic disease
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Original Article Influence of type 2 macrophages (M2) in echinococcosis
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MSC-secreted TGF-β regulates lipopolysaccharide-stimulated macrophage M2-like polarization via the Akt/FoxO1 pathway
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Original Article Status of M1 and M2 type macrophages in keloid
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Original Article Overexpression of IL-12 reverses the phenotype and function of M2 macrophages to M1 macrophages
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Hydroxychloroquine induced lung cancer suppression by enhancing chemo-sensitization and promoting the transition of M2-TAMs to M1-like macrophages
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Interferon-γ and celecoxib inhibit lung-tumor growth through modulating M2/M1 macrophage ratio in the tumor microenvironment
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