• No results found

All strains are available upon request. Supplemental Figures S1-S5 and Supplementary Tables 741

S1-S5 can be found in ‘Supp Figures and Tables.docx’. Supplementary Table S4 contains 742

genotypes for each strain used in this study. Supplementary Table S5 contains nucleotide 743

sequences for each primer used in qRT analysis. RNA Seq gene expression data are available 744

in excel format in file ‘Supplementary Excel Data Set S6.xlsm’ or at GEO with the accession 745

number: xxxxxxx (**submitted, to be assigned) 746

ACKNOWLEDGMENTS: 748

We thank T. K. Blackwell and the Caenorhabditis Genetics Center, which is funded by the NIH 749

National Center for Research Resources (NCRR), for the C. elegans strains. T. K. Blackwell is 750

also acknowledged for sharing an aliquot of the FC4 antibody. We also thank V. Garcia and J. 751

Hourihan for helpful advice regarding the use of phospho-specific antibodies for Western 752

Blotting. The research reported in this publication was supported by the National Institute of 753

Allergy and Infectious Diseases and the National Institute of General Medical Sciences of the 754

National Institutes of Health under award numbers R01AI076406 to DAG and R01GM98200 to 755

SA. SA was also supported by ACS RSG014-044-DDC. A.M.-V. is a member of the EU-ROS 756

(BM1203) and GENiE (BM1408) COST Actions and his work was financed by grants from the 757

Junta de Andalucía (Projects P07-CVI-02697 and P08-CVI-03629). P.S. was supported by the 758

Swedish Research Council, the Torsten Söderberg and Åhlén Foundations, and by the 759

Karolinska Institute Strategic Neurosciences Program. The content is solely the responsibility of 760

the authors and does not necessarily represent the official views of the granting agencies. 761 762 763 764 765 766 767 768 769

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945 946 947 948 949 950 951 ARTICLE SUMMARY 952

The accumulation of free radical damage is an underlying component of numerous human 953

pathologies, ranging from diabetes to aging. This accumulation can be counteracted, however, 954

by an oxidative stress response, which is characterized by the increased production of 955

antioxidants controlled by stress response transcription factors like those of the Nrf family found 956

in mammals. In C. elegans, SKN-1 is a functional ortholog of the Nrf proteins, and this model 957

organism has proven useful to understanding the regulation and the impact of various stress 958

responses. Herein, we demonstrate that intestinal SKN-1 subcellular localization is controlled in 959

part by a thioredoxin, TRX-1, expressed from the distant ASJ neurons, in a manner independent 960

of its redox activity. Interestingly, while the subcellular localization of SKN-1 is affected, 961

activation of this transcription factor, as measured by the expression of SKN-1-dependent genes 962

encoding antioxidants, remains unchanged. This work highlights a novel mechanism of SKN-1 963

regulation in which intestinal localization is impacted in a cell non-autonomous fashion. 964

Moreover, TRX-1-dependent regulation of SKN-1 demonstrates an example in which subcellular 965

localization and transcriptional activation are uncoupled. In conclusion, these findings elucidate 966

another mechanism by which SKN-1 is regulated, adding to the complexity, but exemplifying the 967

importance of tightly regulating stress responses. 968

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