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DERMOID CYST Inspection

o Hyperchloraemic acidosis

EPIDIDYMAL CYST

R: reducability, relationship to each other

11. DERMOID CYST Inspection

Usually single Ovoid / spherical Site: o Congenital, 1-2 cm usually

Along lines of fusion of ophthalmic & maxillary facial processes Inner & outer ends of upper eyebrow

o Acquired, 0.5-1 cm usually

Beneath skin likely to be injured e.g. fingers Scars often present

Palpation

Not warm, maybe tender if infected Smooth surface, Well-defined margins Consistency

o Congenital: Soft (not tense / hard)

o Acquired: Hard & tense (sometimes stony hard) Fluctuant (if large)

Mobile over deeper tissues

o Deep to skin, in subcutaneous tissue

i. Congenital: Not attached to skin or underlying structures ii. Acquired: may be tethered to scar

Background Information

A dermoid cyst is a cyst deep to the skin, lined by skin 2 different methods of formation:

o Congenital: Accident during antenatal development

o Acquired: Implantation of skin into subcutaneous tissue by injury

Clinical features

Congenital (suspect if in child, young adult)

o Formed intra-utero, when skin dermatomes fuse

o Occur at any point in mid-line, common in neck / face / nose

Particularly along lines of fusion of ophthalmic & maxillary facial processes Also: inner & outer ends of upper eyebrow

o May be seen at birth

o Distends a few years later, becomes obvious; few symptoms other than cosmetic problems o Rarely infected

Acquired – Implantation dermoid (suspect if in adult – Browse pg 60)

o Develop when piece of skin survives after being forcibly implanted into subcutaneous tissue Often by injury: cut, stab, etc.

o Symptoms

Small, tense lump

Painful and tender (in areas subjected to repeated trauma) Local effects (e.g. problems with grip / touch if on finger) Also rarely infected

o Differentials

Sebaceous cyst (look for old injury, presence of scar near cyst: more likely dermoid)

Treatment

Congenital

o Surgical treatment; complete excision

o Full extent should first be established with X-ray / CT

Midline cysts may communicate with CSF; must exclude bony defect Acquired

o Complete excision of cyst “Ms.  X  is  a  young  Chinese  girl…”

“On  inspection, there is a single ovoid lump just above the lateral edge of the left eyebrow” “It  measures  3 by 2 cm”

“There  are  no  scars,  ulceration,  or  discharge  seen,  nor  any  overlying  or  surrounding  skin  changes)” “On  palpation, the overlying skin is not warm. It is non-tender.”

“The  surface  is  smooth, with clearly-defined margins” “The  consistency  is  firm, and it is fluctuant”

“The  lump  is  not  attached to the overlying skin nor underlying tissues.” “It  is  fully mobile in  all  directions.”

“My  provisional  diagnosis  is  a  congenital dermoid cyst” “My  differentials  are:  sebaceous  cyst,  lipoma”  

12.

SEBORRHOEIC KERATOSIS

(Senile wart / seborrhoeic wart / verruca senilis / basal cell papilloma)

Inspection

Often multiple

Any part of skin; most found on back & face Round / oval

Light  brown  →  black

“stuck  on  appearance”;;  appears  warty Varying size; Few mm to 2-3 cm Distinct margins

Palpation

No warmth, no tenderness

Rough surface (sometimes papilliferous) More firm than surrounding skin

Attached to skin Special tests

o May be picked off gently – reveals patch of pale-pink skin, 1-2 surface capillaries (bleed slightly) (DON’T  DO  THIS  IN  EXAM)

Request to look for similar lesions elsewhere

Background Information

Benign outgrowth of basal layer of epidermis

o Raised above the level of normal epidermis Microscopy:

o Hyperkeratosis (thickening of keratin layer) o Acanthosis (thickening of prickle cell layer) o Hyperplasia of variably pigmented basaloid cells

Clinical features

Occur in both sexes

More common in elderly people Begin as a patch,

o increases in area, size over months / years o May not increase in thickeness

o May suddenly fall off: leave pale-pink patch of skin Complications:

o May become disfiguring, catch on clothes

o May get infected (may imitate SCC, pyogenic granuloma) o Seldom bleeds (may cause it to change colour to brown)

Leser-Trelat sign: Sudden onset of multiple seborrhoeic keratoses may imply visceral malignancy

Treatment

Non-surgical

o Can be left alone as it is benign Surgical – for cosmetic reasons, etc.

o Superficial shaving (lies above level of normal epidermis) o Cautery

“Mr.  X  is  an elderly  Chinese  gentleman…”

“On  inspection, there is a single ovoid lesion lump on the back” “It  measures  1 by 2 cm”

“The  margins  are  well-defined,  and  it  appears  to  be  slightly  raised  above  the  skin”   “There  are  no  scars,  ulceration,  or  discharge  seen,  nor  any  surrounding  skin  changes” “On  palpation, the overlying skin is not warm. It is non-tender”

“The  surface is rough and greasy; the consistency is firm” “The  lump  arises from the skin”

“My  provisional  diagnosis  is  seborrhoeic  keratosis” “My  differential  diagnosis  is:  pigmented  naevus,  melanoma” “I  would  like  to  complete  my  examination  by…”

“Looking  for  similar  lesions  elsewhere”

13. HAEMANGIOMA

Background Information

Vascular malformations o Types

Capillary: ⅔ of cases, include the cutaneous haemangiomata, telangiectasias Predominantly venous: venous angioma

Deeper levels of subcutaneous tissue, may extend into muscle / joint May have distended veins over the surface of the mass

Empty with pressure, may have bruit Predominantly lymphatic: lymphangioma circumscriptum o Features

Develop as abnormal proliferation of embryonic vascular network Hamartomas

May ulcerate, induce hyperkeratosis in overlying stratum corneum Many forms of cutaneous haemangiomata: (see table)

o Strawberry naevus (cavernous haemangioma) o Port-wine stain (naevus vinosus)

o Spider naevus

o Campbell de Morgan spot

Strawberry Naevus Port-Wine Stain Spider Naevi Cambell de Morgan Spot Inspection

Site (can be anywhere) Head & Neck Lips, face, mucous membranes of mouth, shoulders, neck, buttock Upper torso, head and neck (drainage of SVC) Trunk (bilat) – upper > lower Occasionally on limbs Number May be multiple Usually single Usually multiple Usually multiple

Colour Bright red / dark red Purple-red Bright red Dark red / deep purple

Size Variable; 1-10 cm Variable Variable; few mm 1-3 mm

Edge Well-defined Well-defined – Well-defined

Shape Sessile  →  pedunculated as they grow larger Variable Variable Circular, may be raised

Skin

Changes of ulceration with scabs May have small areas May have dilated subcutaneous veins around lesion – –

Palpation

Surface smooth, pitted epithelium Irregular, covered with Smooth – Smooth

Consistency Soft – – –

Mobility Mobile – – –

Relations Confined to skin – – –

Special Tests

Compressible: pressure squeezes mass, leaves it

collapsed: slowly refills Not pulsatile

– Compressible, fade completely –

Background Information

Age Infants (congenital) Infants (congenital) – Middle-age  →  elderly

Gender ♂  =  ♀ – – –

Symptoms •  Cosmesis •  May  ulcerate,  bleed  on trauma •  Cosmesis •  Bleeding  may  occur – •  Cosmesis •  Non-tender

Pressure •  Collapses  on  pressure •  Diminishes  colour  but  doesn’t  revert to normal •  Branches  fade  when  arteriole  compressed •  Fade  slightly

Remarks Regress spontaneously (few months – 3 years)

•  Extensive, intradermal •  Present  from  birth,  does  not   change in size

•  Sturge-Weber syndrome: facial PWS with corresponding haemangioma in brain – contralateral focal fits •  Found  in  limbs  when  a/w   Klippel-Tranaunay Syndrome

•  Form  of  telangiectasia •  Dilated  skin  arteriole  feeding   small branches (leaving radially) •  Increase  in  number   •  Associated  with  pregnancy,   chronic liver disease (> 5) – request for abdominal examination

•  Formed  by  collection  of  dilated   capillaries fed by single / small cluster of arterioles •  Have  appearance  of  drops  of   sealing wax

•  No  clinical  significance

Pictures

Also

Telangiectasias

o Dilatation of normal capillaries o Can be secondary to irradiation

o Can be part of hereditary haemorrhagic telangiectasia (Osler-Rendu-Weber syndrome) Autosomal dominant disease

Overt and occult haemorrhage can present as haematuria, haematemesis, melaena, epistaxis, iron-deficiency anaemia

Vin rosé patch

o Congenital intradermal vascular abnormality

o Mild dilatation of vessels in sub-papillary dermal plexus o Can occur anywhere, gives skin pale-pink colour

o Associated with other vascular abnormalities (e.g. haemangiomata, AV fistulae, lymphoedema) o Usually not disfiguring