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Development of a Bespoke Saliva Collector Prototype

Chapter 7: General Discussion and Future Directions

436 7.4. Patient-reported outcomes: their value in self-management protocols 440

7.5. Conclusion and Future Directions 443

References

448

Appendix 1

479

Appendix 2

488

Figure 1.1: COPD pathogenesis 12

Figure 1.2: Combined COPD Assessment 26

Figure 1.3: Location of major and minor salivary glands 46

Figure 1.4: Saliva functions and composition 48

Figure 1.5: Lateral flow assay architecture 56

Figure 1.6: Molecular structure of C-Reactive Protein (CRP) 64

Figure 1.7: Molecular structure of Procalcitonin (PCT) 73

Figure 1.8: Molecular structure of Neutrophil Elastase (NE) 67 Figure 2.1: SalivaBio (Salimetrics, USA) passive drool collection device 82

Figure 2.2: UltraSAL-2 passive drool collection device 82

Figure 2.3: A marked 15ml centrifuge tube (Nunc, Denmark) 84

Figure 2.4: Salivary CRP ELISA kit (Salimetrics, USA) 92

Figure 2.5: Typical CRP “line-of-best-fit for a salivary CRP ELISA. 95 Figure 2.6: Salivary CRP recovery with increasing CRP concentrations 104 Figure 2.7: Salivary CRP recovery across increasing dilution 106 Figure 2.8: VIDAS B.R.A.H.M.S PCT kit (bioMérieux, France) 113 Figure 2.9: Comparison between PBS-T and Control levels 120

Figure 2.13: High range salivary PCT recovery in healthy smokers 132

Figure 2.14: Recombinant PCT levels in PBS-T 138

Figure 2.15A-B: Recombinant PCT levels in healthy non-smoker saliva 141 Figure 2.15C: Recombinant PCT levels in healthy non-smoker saliva 142

Figure 2.16A-B: FTIR Spectra 145

Figure 2.16C-D: FTIR Spectra 146

Figure 2.17: PMN Elastase ELISA kit (Immundiagnostik, Germany) 150 Figure 2.18: Typical four parameters logistic regression curve 153

Figure 2.19: Low range spiked saliva NE recovery 164

Figure 2.20: High range spiked saliva NE recovery 166

Figure 2.21: Pilot saliva sampling protocol 176

Figure 2.22: Saliva flow time in COPD patients at fixed increasing fast times 185 Figure 2.23: Salivary CRP levels in saliva at increasing fast time in COPD patients 190 Figure 2.24: Salivary PCT levels at increasing fast time in COPD patients 193 Figure 2.25: Salivary NE levels at increasing fast time in COPD subjects 196

Figure 2.26: Transferrin assay test strip 195

Figure 2.27: Urine reagent strip and colour test 197

Figure 2.28: Refined saliva sampling protocol 209

Figure 2.29: COPD wellbeing score – manual self-assessment diary 212

subjects 224 Figure 3.2: Baseline variability of salivary CRP levels across healthy non-smokers, health

smokers and stable COPD 225

Figure 3.3: Salivary PCT levels from healthy non-smokers, healthy smokers, stable COPD

subjects 227

Figure 3.4: Baseline variability of salivary PCT levels across healthy non-smokers, healthy

smokers and stable COPD 228

Figure 3.5: Salivary NE from healthy non-smokers, healthy smokers and stable COPD subjects 230 Figure 3.6: Baseline variability of salivary NE levels across healthy non-smokers, healthy

smokers and stable COPD 231

Figure 3.7A-B: Correlations of COPD patients’ PRO and salivary biomarkers levels 235 Figure 3.7C: Correlations of COPD patients’ PRO and salivary biomarkers levels 236 Figure 3.8A-B: Salivary biomarkers in COPD exacerbations 240 Figure 3.8C: Salivary biomarkers in COPD exacerbations 241 Figure 3.9A-B: Correlations between same subject serum and salivary biomarkers 243 Figure 3.9C-D: Correlations between same subject serum and salivary biomarkers 244 Figure 3.9E-F: Correlations between same subject serum and salivary biomarkers 245 Figure 4.1A: Electronic Wellbeing and Self-Assessment diary log-in screen shots 261 Figure 4.1B: Electronic Wellbeing and Self-Assessment diary log-in screen shots 262

265

Figure 4.3: Research team structured clinical log 266

Figure 4.4A: The Research team section screen shots 267

Figure 4.4B: The Research team section screen shots 268

Figure 4.5A: Host-website screenshot 286

Figure 4.5B: Host-website screenshot 287

Figure 4.6: Stable and prodromal phase COPD Wellbeing scores 290

Figure 4.7: Exacerbation phase COPD Wellbeing score 293

Figure 4.8: Cumulative Sum chart of COPD Wellbeing score 294 Figure 4.9: Post Exacerbation-Recovery COPD Wellbeing score 296 Figure 4.10: Forced expiratory volume in one second across the 4 defined COPD phases 300 Figure 4.11: Forced expiratory volume in one second clusters in the stable-prodromal period

304 Figure 4.12: Forced vital capacity across the 4 defined COPD phases 308 Figure 4.13: Forced vital capacity cluster across stable-prodromal phases 311 Figure 4.14: Salivary C-reactive protein (CRP) levels across the 4 defined COPD phases in the

whole patient exacerbator group 315

Figure 4.15: Salivary C-reactive protein (CRP) levels across the 4 defined COPD phases in the

patients who experienced raised analyte levels 316

Figure 4.16: ROC curve of salivary CRP levels 318

Figure 4.18: Salivary Procalcitonin (PCT) levels across the 4 defined COPD disease phases in

the whole exacerbator group 328

Figure 4.19: Salivary Procalcitonin (PCT) levels across the 4 defined COPD phases in patients

who experienced a raised analyte levels 329

Figure 4.20: ROC curve of salivary PCT levels 331

Figure 4.21: Salivary PCT clusters in the stable-prodromal phases 335 Figure 4.22: Salivary Neutrophil Elastase (NE) levels across the 4 defined COPD phases in the

whole exacerbator cohort 339

Figure 4.23: Salivary Neutrophil Elastase (NE) levels across the 4 defined COPD phases in

patients who experienced raised analyte levels 340

Figure 4.24: Salivary NE clusters across the stable-prodromal phases 344 Figure 4.25: Factor plot of the 3 exacerbation phenotypes 353

Figure 5.1: Populations and workshop recruitment 368

Figure 6.1: Workshop recruitment 387

Figure 6.2: Ergonomic lip feature 395

Figure 6.3: First (conic) Filter 397

Figure 6.4: Cellulose acetate filter 398

Figure 6.5: 3D-printed prototype saliva collector 400

Figure 6.9: Salivary CRP levels across different saliva processing methods 413 Figure 6.10: Salivary PCT levels across different saliva processing methods 415 Figure 6.11: Salivary NE levels across different saliva processing levels 417 Figure 6.12: Unadulterated compared to collector filtered standard NE levels 421

Table 1.1: 2001 GOLD classification of COPD severity 23 Table 1.2: Updated COPD severity classification GOLD 2003 24

Table 1.3: Modified MRC dyspnoea score 28

Table 1.4: Summary of COPD scoring systems 30

Table 1.5: Technological approaches to salivary analysis 62

Table 2.1: Thesis subjects’ demographics and allocation 89

Table 2.2: Generic salivary CRP ELISA plate layout 94

Table 2.3: COPD patient demographics 97

Table 2.4: Saliva spiking experimental protocol 99

Table 2.5: Saliva dilution experimental protocol 99

Table 2.6: The dilution and recovery experiment protocol for each COPD patients’ sample of

saliva 100

Table 2.7: Salivary CRP levels and recovery across increasing spiked concentrations 103 Table 2.8: Endogenous salivary CRP levels and recovery percentage at increasing dilutions

107 Table 2.9: Manufacturer endogenous salivary CRP levels and recovery percentage at increasing

dilutions 108

Table 2.13: High range PCT spiking experimental protocol 118 Table 2.14: Endogenous PCT levels in saliva from healthy individuals 121 Table 2.15: Low range PCT levels and recovery in healthy non-smokers 123 Table 2.16: High range PCT levels and recovery in healthy non-smokers 126 Table 2.17: Low range PCT levels and recovery in healthy smokers 129 Table 2.18: High range PCT levels and recovery in healthy smokers 131 Table 2.19A: Protocol for PBS-T spiked with recombinant PCT experiment 135 Table 2.19B: Protocol for recombinant PCT neat saliva spike experiment 136

Table 2.20: Recovery of PCT in spiked PBS-T samples 137

Table 2.21: Recombinant PCT recovery in spiked saliva 140

Table 2.22: Generic NE microtitre plate layout 151

Table 2.23: Group demographics 156

Table 2.24: Dilution saliva NE experiment microtitre plate layout 157 Table 2.25: Spiked saliva NE experiment microtitre plate layout 157 Table 2.26: Unadulterated saliva dilution experimental protocol 158 Table 2.27: Low range NE spiking experimental protocol 159 Table 2.28: High range NE spiking experimental protocol 160 Table 2.29: Endogenous salivary NE levels across varying dilutions in all subjects 162

subjects (n = 5) 163 Table 2.31: High range salivary NE levels across varying dilutions in both COPD and healthy

subjects (n = 5) 165

Table 2.32: Demographic details for participating healthy and COPD patients 180 Table 2.33: A comparison of the time taken by all healthy subjects and COPD patients to produce a set volume of saliva (2mls), with associated saliva flow rates 182 Table: 2.34: A comparison of the time taken to produce a set volume of saliva (2mls) at different fast times after eating and associated saliva flow rates in COPD patients 183 Table 2.35: Saliva CRP levels at increasing fast time in COPD patients 189 Table 2.36: Saliva CRP significance levels at increasing fast time in COPD patients 189 Table 2.37: Saliva PCT levels at increasing fast time in all COPD patients 192 Table 2.38: Salivary NE levels at increasing fast time in all COPD patients 194

Table 2.39: Demographic details of COPD patients 204

Table 2.40: Demographic breakdown of all saliva samples tested for blood contamination 206 Table 3.1: Subject demographics and salivary biomarker profiles for the healthy non-smokers,

smokers and stable COPD subjects (n = 107) 222

Table 3.2: Correlations of whole study population (n = 143) symptom scores and sputum

salivary biomarker levels 234 Table 3.4: Same COPD patients in stable and exacerbation phase (n = 36) demographics,

salivary biomarker levels and symptom profiles 238

Table 4.1: Data classification definitions 275

Table 4.2: Total study population baseline and stable phase characteristics 280 Table 4.3: Exacerbation and non-exacerbators cohort characteristics 281 Table 4.4: Total length of pre-exacerbation phase in the exacerbation cohort 282 Table 4.5: Single and re-exacerbator cohort characteristics 284 Table 4.6: COPD Wellbeing Score sub-populations based on stable-prodromal data 291 Table 4.7: Comparison of COPD Wellbeing Scores between single exacerbator and

re-exacerbator patients 297

Table 4.8: FEV1 values across the whole COPD study cohort 301 Table 4.9: LCGA Goodness of fit tests for stable-prodromal FEV1 data 302 Table 4.10: Homogeneity of COPD patient cluster membership for stable-prodromal FEV1 data 302 Table 4.11: Exacerbator COPD patient cluster membership for stable-prodromal FEV1 data

303 Table 4.12: Covariates between the 2 patient exacerbator clusters for FEV1 and control

non-exacerbator cohort 306

Table 4.13: FVC values across the whole study cohort 309

Table 4.15: Spirometry values between single and re-exacerbators 312 Table 4.16: Salivary CRP levels across the whole COPD study cohort 314 Table 4.17: Sensitivity and specificity of exacerbation phase salivary CRP levels 317 Table 4.18: LCGA Goodness of fit tests for stable-prodromal salivary CRP levels 320 Table 4.19: Homogeneity of COPD patients cluster membership for stable-prodromal salivary

CRP levels 320

Table 4.20: Exacerbator COPD patient cluster membership for stable-prodromal salivary CRP

levels 321

Table 4.21: Multinomial regression of covariates between the 3 exacerbator sub-population

clusters for salivary CRP 325

Table 4.22: Covariates comparisons between the 3 patient exacerbator clusters and

non-exacerbator control cohort for salivary CRP 326

Table 4.23: Salivary PCT levels across the whole COPD study cohort 327 Table 4.24: Sensitivity and specificity of exacerbation phase salivary PCT levels 330 Table 4.25: LCGA Goodness of fit tests for stable-prodromal salivary PCT levels 333 Table 4.26: Homogeneity of COPD patients cluster membership for stable-prodromal salivary

PCT levels 333

Table 4.27: COPD patients cluster membership for stable-prodromal salivary PCT levels 334

Table 4.30: LCGA Goodness of fit tests for stable-prodromal salivary NE levels 342 Table 4.31: Homogeneity of COPD patients cluster membership for stable-prodromal salivary

NE levels 342

Table 4.32: COPD patients cluster membership for stable-prodromal salivary NE levels 343 Table 4.33: Multinomial regression of covariates between the 3 exacerbator sub-population

clusters for salivary NE 346

Table 4.34: Covariate comparison between the 3 patient exacerbator clusters for salivary NE

and the non-exacerbator control cohort 347

Table 4.35: Comparison of salivary biomarker levels between single and re-exacerbator patient

cohorts 349

Table 4.36: Multidimensional composite score for each COPD exacerbator patient 351

Table 5.1: Workshop demographics 369

Table 5.2: Workshop questions 371

Table 6.1: Workshop demographics 388

Table 6.2: Workshop questions 389

Table 6.3: Subject demographics 407

Table 6.4: Subject demographics 411

Table 6.5: Endogenous salivary CRP levels: Tube centrifugation compared to modified saliva

collector in the same samples 412

collector in all healthy subjects 414 Table 6.7: Endogenous salivary NE levels: Tube centrifugation compared to modified saliva

collector in the same healthy subjects 416

Table 6.8: Recovery of “standard” NE using two saliva sample processing methods 420

Dedicated to Jaimini Patel (Mum)

“alma mater”

Chapter 1: