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MATERIALS AND METHODS

2.11   Karyotype Analysis

Chromosomes   of   C57BL/6   PαS   MSCs   at   P3   and   P5   were   analysed   for   the   presence   of   any   culture-­‐induced  abnormalities.  The  same  culture  was  maintained  between  both  time  points,   allowing  us  to  identify  whether  any  clonal  growth  of  abnormal  cells  took  place  (Figure  2.4).   This  work  was  done  in  collaboration  with  Ms  Mary  Strachan,  Dr  Sara  Dyer  and  Prof.  Mike   Griffiths  at  the  West  Midlands  Regional  Genetics  Laboratory,  Birmingham  Women’s  Hospital.    

 

Figure  2.4  |  Overview   of   PαS   MSC   karyotyping.  PαS  MSCs  were  grown  in  SM  ±  GFs.  An  aliquot  of  cells  was   taken  for  G-­‐banding  analysis  at  P3  and  the  same  culture  was  maintained  until  P5  when  another  aliquot  was   taken.  This  allows  us  to  detect  any  clonal  expansion  that  could  have  taken  place.    

 

  2.11.1  PαS  MSC  Harvesting  

PαS  MSCs  were  expanded  as  described  previously  in  SM±GFs.  When  cultures  reached  70%   confluency,  a  1:50  dilution  of  B-­‐ONC  (BRDU-­‐overnight  colcemid)  was  added  and  incubated   overnight.  Colcemid  prevents  spindle  fibre  formation  and  arrests  dividing  cells  in  metaphase.  

Fresh   colcemid   was   added   the   morning   after   to   each   flask   and   incubated   for   an   hour.   Samples  were  washed  in  PBS  and  harvested  using  TrypLE  to  yield  a  single  cell  suspension.   Cell  pellets  were  slowly  resuspended  in  a  hypotonic  0.025M  KCL  solution  and  incubated  at   37˚C   for   20   minutes.   This   solution   causes   the   cell   nuclei   to   swell   in   size   to   prevent   the   chromosomes   from   overlapping   each   other.   After   incubation,   samples   were   spun   at   1500rpm  for  5  minutes  and  the  supernatant  discarded.  The  pellet  was  resuspended  in  the   residual  volume  that  remained  and  flicked  gently  to  ensure  a  single-­‐cell  suspension.  Samples   were  then  slowly  fixed  in  Carnoy’s  fixative  (1  part  glacial  acetic  acid  to  3  parts  methanol).   Samples  were  then  spun  at  1500rpm  for  5  minutes  and  further  fixative  added  in  a  drop-­‐by-­‐ drop  basis.  The  supernatant  was  discarded  after  centrifugation  and  sample  tubes  stored  at   4°C  until  slide  making  and  Giemsa  banding  (G-­‐banding).    

 

  2.11.2  Chromosome  Spreads  

The  fixed  cell  suspensions  were  used  to  make  slides  for  cytogenetic  examination.  Fixed  cell   samples  were  aspirated  in  a  pipette  and  held  over  the  middle  of  each  slide  from  a  height  of   approximately  50cm.  One  drop  was  allowed  to  fall  and  hit  the  slide,  whereupon  the  nuclei   would   burst   and   the   chromosomes   would   spread   across   the   slide.   Slides   were   checked   to   ensure  minimal  overlapping  chromosomes  and  the  cell  density  altered  if  necessary  by  adding   more  fixative.  Slides  were  then  ‘aged’  overnight  at  60°C  prior  to  G-­‐banding.    

  2.11.3  Giemsa  Banding,  Imaging  and  Analysis  

Leishman's  stain  (belonging  to  the  same  family  of  stains  as  Giemsa)  was  used  in  this  study  to   mark  chromosomes  for  cytogenetic  analysis.  The  banding  pattern  observed  with  Leishman’s   stain   is   virtually   identical   to   Giemsa   (Moore   and   Best,   2001).   As   such,   both   stains   can   be   interchangeably  used  for  analysis.    

 

‘Aged’   slides   were   allowed   to   cool   to   room   temperature   and   rinsed   gently   in   tap   water.   Slides  were  then  incubated  in  0.25%  trypsin  for  90  seconds  and  then  rinsed  in  saline  solution.   Slides  were  incubated  in  Leishman’s  stain  for  2  minutes  before  being  rinsed  in  excess  saline.   Excess  saline  was  removed  using  blotting  paper  and  the  slides  were  left  on  a  hot  plate  at   60°C  to  air-­‐dry  before  coverslipping.    

 

Each   slide   was   examined   using   a   standard   light   microscope   and   images   taken   when   a   metaphase  spread  contains  all  40  non-­‐overlapping  mouse  chromosomes  in  the  same  optical   field   was   observed.   Individual   chromosomes   were   electronically   cut   and   arranged   to   produce   a   karyogram   where   sister   chromatids   are   arranged   in   order.   A   total   of   60   metaphase  spreads  per  culture  condition  at  P3  and  P5  were  analysed  by  Ms  Mary  Strachan   for  the  presence  of  any  karyotypic  abnormalities.  These  findings  were  independently  verified   by  Dr  Sara  Dyer  before  the  final  karyotype  was  reported.          

2.12  Immunohistochemistry  

2.12.1  Formalin-­‐fixed  Paraffin-­‐embedded  (FFPE)  Tissue  

FFPE   tissue   blocks   were   cut   at   5µM   thickness   and   fixed   onto   microscope   slides   (X-­‐tra   Adhesive,   Leica).   Slides   were   dewaxed   and   rehydrated   by   passing   through   xylene   (3x   2   minutes  each),  IMS  (2x  2  minutes  each)  and  water  (1x  2  minute  wash).  Antigen  retrieval  was   performed  using  a  high  pH  buffer  (Vector  Laboratories,  Peterborough,  UK).  The  buffer  was   pre-­‐warmed  in  a  microwave  for  5  minutes  at  high  power  before  slides  were  added.  It  was   then  heated  for  a  further  15  minutes  before  being  left  to  stand  for  10  minutes.  The  buffer   was  cooled  by  the  gentle  addition  of  cold  tap  water  until  it  reached  room  temperature.      

Slides  were  then  dried  and  sections  were  encircled  using  a  wax  pen.  Endogenous  peroxidase   activity   was   blocked   by   incubating   sections   in   a   pre-­‐diluted   peroxidase   blocking   solution   (Dako,   Ely,   UK)   for   40   minutes.   Slides   were   then   washed   in   Tris-­‐buffered   saline   +   0.1%   Tween20  (TBS-­‐T;  2x  5  minutes  each)  before  blocking  in  1x  casein  buffer  (Vector  Laboratories)   for  60  minutes.  Primary  antibodies  were  diluted  in  TBS  at  the  appropriate  dilutions  (Table   2.6)  and  added  directly  the  after  casein  blocking  stage.  Slides  were  incubated  with  primary   antibody  at  room  temperature  for  60  minutes  with  gentle  rocking.  Unbound  antibody  was   removed  by  two  further  TBS-­‐T  washes  (5  minutes  each).  Sections  were  then  incubated  with   pre-­‐diluted   horseradish   peroxidase   (HRP)-­‐conjugated   secondary   antibody   (Vector   Laboratories)  for  30  minutes  at  room  temperature  with  gentle  rocking.  Unbound  secondary   antibody   was   removed   in   three   TBS-­‐T   washes   (5   minutes   each)   before   chromogenic   visualisation.  

3,3'-­‐Diaminobenzidine   (DAB;   AbD   Serotec,   Kidlington,   UK)   was   prepared   according   to   manufacturer’s   instructions   and   incubated   with   sections   until   brown   staining   was   visible   (typically  around  30-­‐120  seconds).  Sections  with  an  isotype-­‐matched  control  (IMC)  primary   antibody  were  run  at  the  same  time  to  check  for  non-­‐specific  binding.  Excess  substrate  was   washed  off  in  tap  water  for  5  minutes  to  stop  the  chromogenic  reaction.  Sections  were  then   counterstained   in   Mayer’s   haematoxylin   and   “blued”   for   150   seconds   in   tap   water.   Slides   were   then   dehydrated   by   passing   through   IMS   twice   and   xylene   three   times   before   mounting   coverslips   using   DPX.   DPX   was   allowed   to   set   overnight   before   imaging   on   a   conventional  light  microscope  (Carl  Zeiss  AG).      

 

  2.12.2  Snap-­‐Frozen  Tissue  

In  some  cases,  tissue  was  snap-­‐frozen  in  liquid  nitrogen  and  embedded  in  OCT  compound   (Sakura   Tissue-­‐Tek).   7µm   sections   were   cut,   mounted   on   pre-­‐coated   glass   slides   (Thermo   Scientific),  and  fixed  in  acetone  for  10  minutes.  Excess  acetone  was  removed  in  two  washes   of  TBS-­‐T  (5  minutes  each).  Endogenous  peroxidases  were  blocked  by  incubating  slides  for  15   minutes  in  a  pre-­‐diluted  peroxidase  blocking  solution  (Dako).  Sections  were  then  blocked  in   1x   casein   buffer   (Vector   Laboratories)   for   30   minutes   before   primary   antibody   (Table   2.6)   was  added.  Sections  were  left  to  incubate  at  room  temperature  for  60  minutes  with  gentle   rocking.  Unbound  primary  antibody  was  removed  in  two  washes  of  TBS-­‐T  (5  minutes  each)   and  HRP-­‐conjugated  secondary  antibody  (Vector  Laboratories)  was  added  for  30  minutes  at   room   temperature   with   gentle   rocking.   Sections   were   then   washed,   stained   with   DAB,