• No results found

OBSTRUCTIVE DISORDERS

In document PEDIATRIC NURSING Review (NLE) (Page 49-56)

Gastric Motility Disorder:

OBSTRUCTIVE DISORDERS

A. PYLORIC STENOSIS – hypertrophy of muscles of pylorus causing narrowing &

obstruction. 1.) outstanding Sx- projectile vomiting

- vomiting is an initial sx of upper GI obstruction

- vomitus of upper GI can be blood tinged not bile streaked. (with blood) - vomitus of lower GI is bilous ( with pupu)

- projectile vomiting – increase ICP or GI obstruction - abd distension – major sx of lower GIT obst

2.) met alk

3.) failure to gain wt

4.) olive shaped mass – on palpation

5.)serum electrolyte – increase Na & K, decrease chloride 6.) ultrasound

7.) x ray of upper abd with barium swallow reveal “string sign” Mgt:

1. Pyleromyotomy

2. Fredet Ramstedt procedure

INSTUSSUSCEPTION- invagination or telescoping of position of bowel to another

Common site – ilio-secal junction Prone pt: person who eats fat

Complication – peritonitis – emergency Sx:

1.) persistent paroxysmal abd pain 2.) vomiting

3.) currant jelly stool- dye bleeding & inflammation - palpate sausage shaped mass

Mgt:

1.) Hydrostatic reduction with barium enema 2.) Anastomosis & pull thru procedura

Inborn Errors of Metabolism- deficient liver enzymes

PHENYLKETONURIA (PKU) – deficiency of liver enzymes (PHT)

Phenylalaninehydroxylase Transferase – liver enzyme that converts CHON to amino acid 9 amino acids:

valine isolensine tryptophase lysine phenylalanine

1.) fair complexion 2.) blond hair 3.) blue eyes

Thyroxine – decrease basal metabolism - accumulation of Phenyl Pyruvic acid 4.) Atopic dermatitis

5.) musty / mousy odor urine 6.) seizure – mental retardation

Test – GUTHRIE TEST – specimen – blood - preparation increase CHON intake - test if CHON will convert to amino acid specimen and urine

mixed with pheric chloride, presence of green spots at diaper a sign of PKU

DIET:

Low phenylalanine diet- food contraindicated- meats, chicken, milk, legumes, cheese, peanuts

Give Lofenalac- milk with synthetic protein

Galactosemia – deficiency of liver enzyme

- GUPT – Galactose Urovil Phosphatetranferase - Converts galactose to phosphate tranferace glucose

Galactose – will destroy brain cells if untreated – death within 3 days Dx:

Beutler test – get blood -done after 1st feeding presence of glucose in blood – sign of galactosemia galactose free diet lifetime

neutramigen – milk formula

CELIAC DISEASE – gluten enteropathy

Common gluten food: Intolerance to food with brow B- barley

R- rye O- oat W- wheat

Early Sx:

1. diarrhea – failure to gain wt ff diarrheal episodes 2. constipation

3. vomiting Late Sx:

1. abd pain – protruberant abd even if with muscle wasting 2. steatorrhea

Celiac Crisis- exaggerated vomiting with bowel inflammation Dx:

1. lab studies – stool analysis

2. serum antiglyadin – confirmatory of disease gluten free diet – lifetime

all BROW – not allowed ok – rice & corn

Mgt:

1. vitamin supplements 2. mineral supplements 3. steroids

POISONING- common in toddlers. (falls- common to infant)

1. determine substance taken, assess LOC

Gluten – glutamine ( normal absorption)

Gliadin ( toxic to epithelial cells of villi of intestines, effects is malabsorption syndrome)

Malabsorption

Fats CHON & CHO

peripheral edema & malnutrition

Vit D calcium Vit K Iron folic acid

Inadequate blood

coagulation

Steatorrhea Osteomalasia Bleedin

gg

2. unless poison is corrosive, caustic (strong alkali such as lye) or a hydrocarbon, vomiting is the most effective way to remove poison.

- Give syrup 1 pecac to induce vomiting 3. 1 pecac – oral emetic

- 15 ml – adolescent, school age & pre school - 10 ml to infant

4. UNIVERSAL ANTIDOTE- charcoal, milk of magnesia & burned toast 5. Never adm charcoal before 1 pecac

6. antidote for acetaminophen poisoning – acetylsysterine ( mucomyst)

7. caustic poisoning ( muriatic acid ) neutralize acid by giving vinegar . Don’t vomit prepare tracheostomy set

8. Gas- mineral oil will coat intestine

Lead poisoning

Lead = Destroy RBC functioning = Hypochornic Microcytic Anemia = Destroy kidney functioning

Accumulation of anemia = Encepalopathy Sx:

1. beginning sx of lethargy

2. impulsiveness, learning difficulties

3. as lead increases, severe encepalopathy with seizure and permanent mental retardation Dx: 1. Blood smear 2. abd x ray 3. long bones Mgt:

1. remove child from source

2. if > 20 ug/dL – need chelation therapy = binds with led & excreted by kidney =nephrotoxic

Amogenital

Female:

Pseudomenstration slight bleeding on vagina related to hormonal changes Tearing of fourchette with blood – rape/ child abuse

Rape- Report within 48 h

Shape pubic hair in inverted triangle ( female) Male:

Undescended testes – cyrptorchidism -common to preterm surgery – orchidopexy

baby – pee within 24 h -check for arch of urination

Epispadias- urinary meatus located dorsal or above glans penis Hypospadias- urinary meauts loc ventral or below glans penis

Hypospadias with chordee- fibrous band causing penis to curb downward Mgt:

Surgery

Phimosis- tight foreskin

Balanitis-infection of glands penis – due smegma Mgt:

Circusicion

Hydroseal – fld filled scrotum

Tst of Dx:

Transillumination with use of flashlight - glowing sign

Varicoseal – enlarged vein of epididimis ( girls- vulvular varicosities)

Renal Disorder Cause Sx Tx NSG CARE

NEPHROTIC SYNDROME

infectious 1. Anasarca- gen edema 2. massive protenuria 3. microscopic or no hematuria 4. serum CHON decreased 5. serum lipid increased 6. fatigue 7. normal or decreased BP Prednisone Diuretic Focus of care: monitor edema - weigh daily Diet: Increase CHON Increase K- OJ, beef broth, banana Decrease Na AGN ( acute Glomerulo Nephritis) 3A’s; AGN, autoimmune, Autoimmune Grp A beta hemolytic streptococcus 1. (PPP) primary peripheral periobital edema 2. moderate protenuria 3. gross hematuria 1. anti HPN drug - hydralazine or apresoline 2. iron 1. weigh daily 2. monitor BP & neurologiuc status 3. Diet: decrease K, decrease Na

4. serum K increased 5. fatigue 6. increase BP Complication : 1. hypersensive encephalopathy 2. anemia

BACK- check for flatness & symmetry

Open Neural Tube Defect- decreased Folic Acid intake

SPINA BIFIDA OCCULTA- failure of post laminae of vertebrae to fuse Sx: dimpling of back , Abnormal tufts of hair

SPINA BIFIDA CYSTICA- failure of post laminae of vertebrae to fuse with a sac

Types:

1. Meningocele – protrusion of CSF & Meninges

2. Myelomeningocele – protrusion of CSF & Meninges & spinal cord ( most dangerous)

3. Encephalocele ( CNS complication – hydrocephalus) – cranial meningocele or myelomeningocele

Most common problem - rupture of sac - prone pos

- sterile wet dressing

Most common complication - infection

Myelomeningocele – genitourinary complication- urinary & fecal incontinence Nsg care: always check diaper

Orthopedic complication – paralysis of lower extremities Surgery to prevent infection

Post op – prone position

SCOLIOSIS- lateral curvature of the spine

2 types:

1. structural – rye neck

2. postural – improper posture Dx:

1. uneven hemline

2. bend forward- 1 hip higher

1 shoulder blade more prominent Nsg care:

1. conservative – avoid obesity, exercise

3. corrective surgery – insert Harrington rod post op- how to move

log rolling- move client as 1 unit

EXTREMITIES:

check # of digits = 20

1. syndactyly – webbing of digits 2. polydactyly – extra digits 3. olidactyly – lack of digits

4. Amelia – total absence of digits

5. pocoamelia- absence of distal part of extremities

ErQ duchennes – paralysis- brachial plexus injury or brachial palsy

- birth injury caused by lateral & excessive traction during a breech injury Sx:

1. unable to abduct arms from shoulders, rotate arm externally or supinate forearm 2. absence or asymetrical moro reflex

Mgt:

1. abduct arm from shoulders with elbow flex.

CONGENITAL HIP DISLOCATION – head of femur is outside acetabulum Types;

1. subluxated – most common type 2. dislocated

Sx:

1. shortening of affected leg 2. asymmetrical gluteal fold 3. limited movement – earliest sx

4. (+) ortolanis sign – abnormal clicking sound

5. when able to walk – child limps – late sx- trendelenburg sign Goal of Mgt:

Facilitate abduction Mgt.

1. triple diaper 2. carry baby astride 3. Frejka splint 4. Pavlik harness 5. Hip Spica Cast

TALIPES – “clubfoot”

a.) Equinos – plantar flexion – horsefoot

b.) Calcaneous – dorsiflexion – heal lower that foot anterior posterior of foot flexed towards anterior leg

Equino varus- most common Assessment:

1. Straighten legs & flexing them at midline pos Mgt:

1. Corrective shoe- Dennis brown shoe, spica cast Fx: of cast –

- to immobilize - bone alignment - prevent muscle spasm

lead pencil – mark area to be amputated cold H20 – hasten setting process

hot H20- slow setting process

After cast application – how to move pt:

- use open palm not fingers- fingers will cause indention - dry cast – natural air not blower

- priority check : neurovascular check C- circulation

M- motion S- sensation Cast – with bleeding

- mask with ball pen edge of blood to know if bleeding is on going sign cast is dry = resonant sound, cast cold to touch

-do petaline – making rough surface of cast smooth

CRUTCHES

Fx: To maintain balance - To support weakened leg

Principles in crutches

- wt of body on palm!

- Brachial pulsing – if wt of body in axila - Do palm exercise- squeeze ball

Different crutch Gaits:

1. Swing Through 2. Swing to

- no weight bearing are allowed into lower ext 3. Three point Gait

- wt bearing is allowed in 1 ext 4. Four point gait

5. Two point Gait

In document PEDIATRIC NURSING Review (NLE) (Page 49-56)

Related documents