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Depletion of the cdk Inhibitor p16INK4a Differentially Affects Proliferation of Established Cervical Carcinoma Cells

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Academic year: 2019

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Figure

FIG 1 Knockdown of p16fields from three independent experiments. Two representative micrographs are also shown
FIG 2 Knockdown of p16gene in HeLa cells (A) Left panel, HeLa cells were transfected with p16panel, HeLa cells were transfected with p16INK4A leads to reduced expression of the E7 onco-INK4AsiRNA or control siRNA (200 pmol per well) for three times in tota
FIG 3 p16day 4, and day 8). Left panel, at day 8 RNA was prepared and the mRNA levels for p16(meanINK4A knockdown reduces E7 levels in CaSki and MS-751 cells
FIG 4 p16Lower panel, at day 11, cells were assessed for presence of SA-INK4A knockdown does not affect cell proliferation in CaSki andMS-751 cells

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